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An increase in oxidative and endoplasmic reticulum stress is observed in the brains of patients with AD, and these factors may contribute to neuronal cell death and cognitive dysfunction

If excess acetyl CoA is created and overloads the capacity of the Krebs cycle, the acetyl CoA can be used to synthesize ketone bodies

Those seizures are the result of the interaction between pathological excitation and a lack of inhibition in the neuronal networks of the CNS

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Phosphorylation bar-coding of free fatty acid receptor 2 is generated in a tissue-specific manner